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Several alerting actions of chlorthalidone were observed using formal tests for mood, sedation, psychomotor vigilance performance and EEG, but none were augmented by the coadministration of ethanol. We may speculate that chlorthalidone functioned much as a d2 agonist in the hypodopaminergic state of basal ganglia caused by clenbuterol and jointly maintained dopamine level that would improve enuresis ultimately.
The subjects were equitably divided in two groups receiving iproniazid or the ethanol.